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STUDY OF RELATION BETWEEN HYPERTENSIVE LEFT VENTRICULAR HYPERTROPHY AND NEUROCRINE FACTORS
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GUO Hang-yuan, LU Duan. STUDY OF RELATION BETWEEN HYPERTENSIVE LEFT VENTRICULAR HYPERTROPHY AND NEUROCRINE FACTORS[J]. Journal of Zhejiang University Science A, 2000, 1(1): 97-98.
@article{title="STUDY OF RELATION BETWEEN HYPERTENSIVE LEFT VENTRICULAR HYPERTROPHY AND NEUROCRINE FACTORS",
author="GUO Hang-yuan, LU Duan",
journal="Journal of Zhejiang University Science A",
volume="1",
number="1",
pages="97-98",
year="2000",
publisher="Zhejiang University Press & Springer",
doi="10.1631/jzus.2000.0097"
}
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%T STUDY OF RELATION BETWEEN HYPERTENSIVE LEFT VENTRICULAR HYPERTROPHY AND NEUROCRINE FACTORS
%A GUO Hang-yuan
%A LU Duan
%J Journal of Zhejiang University SCIENCE A
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%P 97-98
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%I Zhejiang University Press & Springer
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T1 - STUDY OF RELATION BETWEEN HYPERTENSIVE LEFT VENTRICULAR HYPERTROPHY AND NEUROCRINE FACTORS
A1 - GUO Hang-yuan
A1 - LU Duan
J0 - Journal of Zhejiang University Science A
VL - 1
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SP - 97
EP - 98
%@ 1869-1951
Y1 - 2000
PB - Zhejiang University Press & Springer
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DOI - 10.1631/jzus.2000.0097
Abstract: In this study of the relationship between hypertensive left ventricular hypertrophy(LVH) and neurohumor factors and ventricular arrhythmias, 180 cases were divided at random into 3 groups: 60 cases with primary hypertension and LVH(PH+LVH); 60 cases with simple hypertension(PH), and with other diseases and LVH(NPH+LVH). The results showed that 1. The excitability of the sympathetic nerve was not elevated while the activity of the vagus nerve was not significantly decreased in the patients with PH+LVH.The increased sympathetic nervous tension was correlated with the ventricular premature beat which was statistically correlated with the myocardial ischemia. 2. The patients' condition in group PH+LVH was not correlated with the levels of aldosterone and insulin and was not even positively correlated with the levels of renin and angiotensin-II. 3. The patients in group PH+LVH had high incidence of ventricular premature beat and myocardial ischemia. 4. The patients' condition in group NPH+LVH was not even positively correlated with the neurohumor factors but their heart failure was positively correlated with the levels of aldosterone, sympathetic nervous tension, myocardial ischemia and ventricular premature beat.
[1]Francis, G.S., Carlyle, W. C., 1993. Hyperthetical pathways of cardiac myocyte hypertrophy:response to myocardial injury. Eur Heart J, 14(suppl):49.
[2]Grossman, Z., Alster, Y., Shemesh, J. et al. 1994. Left ventricular mass in hypertension: Correlation with casual, exercise and ambulatory blood pressure. J Hum Hypertens, 8(10):741.
[3]Mayet, J., Shahi, M., Poulter, N.R. et al., 1995. Ventricular arrhythmias in hypertension:In which patients do they occur. J Hypertens, 13(2):269.
[4]Perloff, D., 1994. Retrospective and prospective research on hypertension related end-organ damage.J Cardiovasc Pharmacol, 24(suppl.A):SJ-5.
[5]Siche, J.P., Schwebel, C., Longere, P. et al., 1994. Left ventricular hypertrophy and blood pressure variability during rest and ambulatory monitoring in the hypertension patient. Archmal, 32(8):1305.
[6]Zhang, W.Z., Gong, L.S., 1991. Clinical evaluation of the loss of nocturnal decline of blood pressure in patients with hypertension. Chin Med Sci J, 6(suppl):34(in chinese with English abstract).
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